Cat

Reflections Back on 2017 from the New Year

/ Dr. Christine McFadden / Leave a comment

I can’t speak for you, but let me start this by saying that I spent my first night of the new year sleeping on the floor. No, you too? So let our first resolution be to stay home next year! Nothing stronger than water had crossed my lips, we missed the ball drop in New York because we were arguing too much as we channel-surfed at the last minute and I couldn’t tell you if there were midnight fireworks because we were all asleep long before the year crossed over. Some of us on the floor. I was there mostly because the makeshift mattress set up for the kids was very narrow. If you were next to your mom you would be snuggling, but if forced to share the same space with your sister it would have been a war zone all night. I chose “some sleep” over “none”. The great thing to come out of this? Everything settled to the down side and I’m convinced my silhouette, as seen from the left, is much slimmer.

I had lots of time to think while performing this balancing act. I remembered the faces of some I’d said goodbye to in the previous year. “Nella” was a gallant Golden Retriever. He was named as a young dog by a child too young to know a girl name from a boys and owned by a war veteran too kind to correct the child. Nella was the color of a vanilla wafer cookie. He was a big-hearted dog, always gently greeting me with a wave of his plumed tail, no matter the exams, the blood tests or x-rays that were needed. At 16 years of age, he suffered from severe arthritis. His owner always had a kind smile and a silly joke when he came in. He thanked us for our efforts to keep the big dog comfortable. He cooked for Nella when needed. On the bad days we hugged. And finally there was no denying that the most loving thing he could do for his dog was to let him go.

I started 2017 with a C-section delivery on Jan. 1. Ring in the new!

I ended 2017 with an 18 pound cat, Shrek, that had his bladder obstructed by tiny bladder stones they call “sand.” He couldn’t urinate. His bladder filled up tight as an over-filled balloon and the plumbing backed up until the pressure shut down his kidneys. It is hard to walk when your bladder feels like a large boulder in your stomach. When your kidneys shut down you feel tremendous nausea. It’s called uremic poisoning, because the body waste that the kidneys should filter out are reabsorbed into the bloodstream, poisoning you. It is impossible to eat when your kidneys shut down. A true emergency, his bladder at risk of rupture, we rushed Shrek into surgery. Under anesthesia we spent long minutes trying to break down the plug of small grit blocking his urethra until we could place a bladder catheter and empty his bladder. Then we pumped him full of intravenous fluids to encourage his kidneys to go back to work. Shrek looked a little odd, with one tube dripping fluids in at the front end and another dripping out more fluids (urine) at the back end. Key to prevent recurrent formation of this “sand” in Shrek will be his conversion to a diet of food developed specifically to prevent urinary sand of Feline Lower Urinary Tract Disease (FLUTD) syndrome.

My final thoughts before drifting off to sleep were of the first stanza of a poem I memorized years ago. The poem is by Alfred, Lord Tennyson.

Ring out, wild bells, to the wild sky,

The flying cloud, the frosty light:

The year is dying in the night;

Ring out, wild bells, and let him die.

 

May the new year be kind to you and your pets.

Christine B. McFadden, DVM

drmc@mcmenagerie.com

Heatstroke

/ Dr. Christine McFadden / Leave a comment

Dr Mc Blog

 

May 24, 2016
Summer is coming. In Merced that can mean baking temperatures over 100 degrees. Even if the day doesn’t go over the 90’s, the black asphalt paths that line Bear Creek, Black Rascal Creek and many other favorite walking paths will heat up significantly, holding the heat well into the evening hours. People walking in rubber soled sneakers may not notice how hot the ground feels. I actually recommend that clients who take their pets out for an evening stroll bend down and place a flattened palm to the sidewalk or path – only then will they recognize what their dogs’ feet are feeling. Sometimes my hand has burned at 9 o’clock at night! If at all possible, consider early morning walks with your dog – they can be quite refreshing and you might take in an egret or hawk flying overhead.

Dogs are digitigrade (love words!), meaning they stand and bear their weight on their toes, not a sole or heel like a person. Each toe is covered by a protective pad of keratin, a horned callus that protects them from injury. Inside that is thick fatty tissue, a good shock absorber. This pad is soft in young puppies, thickening and hardening with the use they get from exercise and exposure to different terrain. A dog that has sat on the couch all summer will have tender feet if suddenly expected to go game hunting for miles in the fall. That bicycle ride you took, with your dog running gamely behind? Don’t be surprised if he or she comes up lame the next day, bare patches of pad rubbed away to expose bleeding sores. This goes way beyond blisters and will take weeks to heal, as the lost tissue can’t be sewn back on, but must slowly granulate in from the sides of the wound. Walking on soft grass is about all those dogs can bear to do for some time. Don’t take your dog’s feet or athleticism for granted – they need regular, planned exercise for peak performance and health. This is your chance to shine as a Personal Trainer! Don’t blow it!

I would like to believe everyone knows not to leave a dog in a car in a parking lot during our summers. Forget cracking the windows. A CRACKED WINDOW WILL NOT PREVENT HEAT BUILD UP IN THE CAR. It has been estimated that even on a day when the outdoors temperature is in the mid-70’s, that the interior of a car can rise to over 100 degrees or higher in less than 30 minutes. Seventy degrees? Can you imagine how quickly that temperature spikes in our area on a real summer day? One estimate I read stated that on a 90 degree day the inside of a car could reach 110 degrees in less than 10 minutes! The American Veterinary Medical Association (AVMA) posts some interesting and horrible facts from several studies on Pets in Vehicles.  The car acts like a greenhouse, with the air unable to circulate or get back out through the windows. The pet cannot breathe in the overheated air and dies from heatstroke. The dogs core (inside) body temperature may go over 104 degrees. The highest temperature I ever recorded in the stomach of a dog that had died from heatstroke was 110 degrees – our thermometer didn’t register any higher. It was too hot to keep my gloved hand inside to perform the necropsy. (The word “autopsy” is reserved for post-mortem examinations of humans or “self”. An autopsy is an examination done to determine the cause of death. In animals we most commonly use the word necropsy.). These are very sad moments, indeed. California does have laws restricting leaving pets confined inside a car under conditions that endanger the pet’s life. Please, leave your pet at home while you run errands. Here in the Valley make sure your outdoor kennels are not all concrete, baking in the sun. Shade cloth is not enough protection, either. Mature trees, watered and shaded grass, even outdoor misters should all be considered for your dog’s health and comfort during the summer days when they are outdoors. As we see each summer, it is a matter of life or death.

Christine B. McFadden, DVM
drmc@mcmenagerie.com

 

Variety Is The Spice Of Life

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February 17, 2016
They say variety is the spice of life. I am fortunate to have an extremely interesting career! In the past eight days I have seen one rabbit with an ear ache and spayed another. A lizard presented with scoliosis, curvature of the spine, caused by underlying malnutrition. I saw a cat with a very odd skin mite that may not have been diagnosed in Merced before! By the simple act of pressing scotch tape over the cat’s dander and looking at this under the microscope I found a mite I’d never seen before. My colleague, Dr. Klingborg, identified it from a derm (skin) meeting he’d attended in November and now we’ve held several discussions on the presence of this mite in Merced (it is considered to be contagious between cats). And always, the “usual” mix of cats, dogs, and birds. Today while performing spay surgeries (an ovariohysterectomy surgery) on cats I opened up one female cat only to find parts of a mummified fetus in her abdomen, causing no known distress to her before today. I completed the surgery and explained the situation to her owner, using digital photos taken during the surgery. Another veterinarian in the practice, Dr. Lauppe, was doing an orthopedic surgery on an injured knee while Dr. K removed a cancerous mass and attended to a dog that had been attacked by another dog (BBBD – Bit By Big Dog). A guinea pig with an eye ulcer came in and the Zoo needs a review on the Mountain Lion’s diet. A delightful Labrador had five enormous rotting teeth extracted and went home on antibiotics and painkillers. In two weeks he’ll be soooo much better! A lady called about a duck she thought might have eaten a tiny battery (the battery could corrode the stomach and cause heavy metal poisoning. All waterfowl are at risk of ingesting odd metal objects – they just like shiny objects). A woman (I never got her name) brought in a hummingbird that “fell from the sky”. We mixed up sugar water, revived the uninjured bird and sent it off with her to be released where she found it. An abuse case was brought in by Animal Control and a pregnant dog with three puppies stuck inside presented for a Caesarean section, performed beautifully by one of the other vets. I saw a Sulcata African tortoise with a mild upper respiratory disease and discussed the special dietary needs the new owner would want to address. Obtained from a Tortoise rescue group, the Sulcata’s shell mounded up on top, called pyramiding of the carapace. Much like the lizard with the deformed spine, it indicated an improper calcium and vitamin D ratio, the tip of the iceberg for signs indicating malnutrition.

February is National Dental Month for Dogs and Cats and most veterinarians who care for companion animals focus special attention on dental care this month. In veterinary medicine we are not focused on the whitest smile to impress someone! You are most fortunate if you have never experienced a tooth ache. Some dogs present with teeth so brown with mineralized plaque and calculus that it looks like moss has grown on them. That bad breath smell is from the bacteria growing along and under the gum line. The same bacteria that can dislodge and spread to the heart or kidneys. Cats demonstrate “jaw chatter” if you touch a tooth with a neck lesion cavity near the gum line – you can observe this incredibly painful response after you peel your cat off the ceiling. Most dogs and cats continue to eat despite foul teeth – please do not judge your pet’s dental pain by whether they haven’t starved to death! Over the years I have heard countless times of pets that “acted years younger” once their abscessed teeth were removed, their mouths healthy again. Call your vet today – there are some great deals out there this month!

Christine B. McFadden, DVM
drmc@mcmenagerie.com

Feline Urinary Syndrome or FUS, FIC, FLUTD

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Dr Mc Blog

 

December 21, 2015
I think he was already 90 when we first met. The blue eyes, the felt hat reminded me of my own grandfather. A little shorter than me, he was a kind man who had moved to the States when his family emigrated from the Azores. It had been a long time since he worked on a dairy, and his companion now was a black and white cat, Jimmy, vividly marked like a Holstein cow. On that first meeting, Jimmy was just in for a check-up and vaccinations. We talked more about Mr. B’s wife, who had passed away after eating watermelon. He blamed himself for letting her eat the seeds – she loved watermelon so much that she’d inhale chunks heedless of any seed that went with it. He knew the seeds had killed her. I suspected cardiac arrest was the underlying cause from the rest of our discussion. That afternoon we forged a bond that grew over the years. Jimmy’s owner lived independently in a small house in the country. When he could no longer drive a car he rode a three wheeled bicycle, humming along the back roads for miles at a time.

Jimmy came and went as he pleased, growing a little plump under the tender loving care of home, plied with a variety of foods on a near daily basis. One morning Mr.B brought him in, both quite agitated. Jimmy was having trouble walking, crying out, and Mr. B said he kept trying to pee at home. He didn’t think anything was coming out.

When I placed my hand over the cat’s abdomen I immediately felt a hard ball inside, about the size of a baseball. Just that gentle touch yielded a scream of anguish from Jimmy that made us all jump. That ball was the cat’s bladder, swollen near to bursting because the outlet was obstructed. The tube that carries urine from the bladder to the outside is called the urethra. Tiny bladder crystals or struvite, sometimes called “sand” had formed in Jimmy’s bladder. The crystals precipitated from a combination of minerals found in his food, often magnesium, ammonium, phospate. Thirty years ago we called this the Feline Urinary Syndrome. We blamed the diet – no one knew why, or which cats – and knew it could occur in both male or female cats. The crystals irritated the lining of the bladder and urethra, causing swelling, thickening, and pain when urinating. In male cats, the urethra is longer and narrows at the tip, so they can obstruct. If a big clump of “sand” passes at one time it acts like a cork, stopping up the bladder so no urine can pass. Of course, the kidneys are the last to know, so they stay on the job making urine, which fills up the bladder until it can’t get any bigger (or it ruptures). Everything comes to a screeching halt and the kidneys start to shut down (post-renal kidney failure). At this point the cat has a relatively short time to get help – under 24 hours – before it dies.

Treatment at the veterinary hospital is immediate; this is not a procedure that can be safely put off until lunch. Risks are high. The cat must be anesthetized and a urinary catheter threaded carefully backwards up the urethra (retrograde) using a sterile flush if necessary to break down the obstruction. The catheter is placed directly into the bladder and sewn in place, often for 1 or more days. An intravenous catheter is also placed to immediately flush the system and get the kidneys up and running. Blood tests tell the picture of the degree of kidney disease. The cat will have more blood tests before coming off IV’s to be certain that kidney function has been regained. Oddly, most of these cases are not infected with bacteria, though we use antibiotics to prevent a secondary infection once an indwelling bladder catheter is in place. Other names for this syndrome now include Feline Interstitial Cystitis and Feline Lower Urinary Tract Disease.

A change in diet may prevent recurrence of crystal formation. This requires a Prescription diet from your veterinarian. The food maintains the urine PH in a very narrow range that prevents formation of more crystals. In dire circumstances when a cat obstructs a second time, a surgery called a perineal urethrostomy (big words!) is life-saving. The surgery widens the urethra 3-4x more than before. The cat may still form crystals, but it shouldn’t be able to obstruct again.

Jimmy responded well to treatment and readily accepted his new diet. He died from other causes about 2 years later. His owner grieved. It was springtime, and Mr. B had his door open to let in the fresh breeze. He dozed off in his recliner and was startled upon awakening to find a black and white cat, boldly patterned, winding herself through his legs and around the coffee table. He knew it wasn’t Jimmy, but? He named her Susie. She had come to stay. Every time I saw them together we would marvel at how Jimmy must have sent her.

FUS=feline urinary syndrome;FIC=feline interstitial cystitis;FLUTD=feline lower urinary tract disorder. If you like words.

Christine B. McFadden, DVM
drmc@mcmenagerie.com

 

Cat Scratch Fever

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Dr Mc Blog

July 9, 2015
My mother wanted more articles on cats, but this time she’s gone too far. While her cats swirled about her feet in a breakfast feeding frenzy one nudged her along a little too hard, breaking skin. Three days later she almost landed in the hospital as her leg swelled up, an angry red color.

Diagnosis : Cat Scratch Fever.

Cat Scratch Fever, more properly Cat Scratch Disease, is caused by a little known bacteria, Bartonella henselae. Because cats themselves rarely suffer any effects from this bacteria, your veterinarian is not “looking” for it. Oddly enough, statistics from the Center for Disease Control (CDC) in Georgia report that up to 40 % of cats may harbor this bacteria on their claws or teeth sometime during their life, with the most likely period being when they are kittens. This, of course, is when young cats are most playful, most likely to play bite or scratch during a wrestling match. If your skin is broken the Bartonella bacteria has an opportunity to start an infection in you – the human. The cat will be fine.

Even more oddly, your cat probably picked the bacteria up when it was scratching at a flea! Infected fleas carry the bacteria, and your cat will get flea dirt caught under its nails or in its’ teeth. Once a person’s skin is broken, the bacteria is transmitted through the saliva or from under the cats nail. Even allowing a cat to lick an open wound which you may have (irritated mosquito bite?) may transfer the bacteria. We’re back to flea control, again! In our mild climate, fleas are a reality year round in Merced. If you think you have them under control, keep treating – keep it that way!

Washing any scratch or bite wound will prevent most people from developing an infection. Once Bartonella has taken hold, it can be readily treated with antibiotics by your physician. Your cat does not require treatment. On rare occasion an infection with Bartonella can land a person in the hospital for intensive care.

Does Bartonella EVER bother the cat? Maybe. There are tests to demonstrate if anti-Bartonella antibodies are present in the blood stream. This only proves exposure to the bacteria, not current infection. Rarely an ill cat, perhaps with an eye infection, may be tested for Bartonella. If you have high concern about acquiring a new cat or are faced with a family member who is immunocompromised and must guard against any disease encounter you can request these tests for your cat and even a course of treatment as a preventative, though it can’t guarantee against future re-infections.

My mothers’ cats are on flea control and are older, but one of them goes outdoors and inevitably brings fleas back into the house. I knew the source of infection and how to contain it. I must admit it was interesting to follow how her physician handled it. As a veterinarian, I am familiar with the rejection one must inevitably face when serving in a “helping profession”. Everyone wants to help. They want to help even more when it’s outside their scope of expertise – it’s so tempting to give advice! I am sometimes the last stop in a desperate quest to heal a pet, after the owner has purchased a variety of compelling exotic potions over-the-counter or whipped up a guaranteed home remedy recommended by the neighbor’s cousin passing through town.  My mothers’ leg improved slowly but surely under the extremely watchful eye of her physician (he even offered to make a house call to check on her over the weekend). So it was with some interest as I followed her recovery to hear her inform me that she was going to have the gardener check her leg that day. That gave me pause. I double-checked. The gardener? I silently chewed this over in my mind. She was seeing her physician every other day, if not daily. Her veterinarian daughter was stopping by every night. The gardener?  It felt good to know these things didn’t happen just to me. I wondered if the gardener would argue the merits of herbal remedies….? I could tell this was a turning point for the better in her case and that I could step out now. And her physician? A miraculous cure. My hat’s off to you, Dr. De La Pena.

Christine B. McFadden, DVM
drmc@mcmenagerie.com
Valley Animal Hospital, Merced

Miliary Eczema in Cats

/ Dr. Christine McFadden / Leave a comment

 

Dr Mc Blog

May 27, 2015
My mother has complained of a dearth of articles about cats. Here you are, Mom. The domestic feline or house cat is believed to have appeared as a companion as early as 7,000 or more years BC. Whether part of ancient religions or welcomed as a mouse catcher, the cat was also made to be a pet. Although today many argue the value of a cat compared to a dog, statistics indicate that there are as many, or more pet cats in the world as dogs.

Cats are athletic, inquisitive, intelligent and independent.

One such special cat was Alfred. A sleek golden orange tabby, he had grown into a magnificent 12 pound, well-muscled  cat. Alfred ran his house, assuring punctual meals with scheduled outdoor exercise and afternoon naps in between.  He was about three years old when his owner noticed that his coat was looking a little patchy, and when she picked him up to pet him she felt tiny scabs over his back. Alfred seemed a little irritable when she tried to pet him, squirming away as if his skin hurt. This would not do, so off Alfred and owner went to the vet!

Close examination revealed that Alfred had scratched at himself, evidence of itchy, or pruritic skin. The fine little scabs were clustered close together over his skin, with a few slightly bald patches where he must have scratched off some fur. The working diagnosis for a cat with this kind of skin condition or dermatopathy is called “miliary eczema”. “Miliary” meaning “a rash that resembles tiny seeds” and “eczema” meaning inflamed skin, often itchy or red. This is sort of a catch-all term that describes the rash but not necessarily the underlying cause.

Most often a rash might occur in response to an allergy. But allergy to what? In Alfred’s case his owner medicated him monthly with a flea preventative and indeed, there was no evidence of flea “dirt” (blood sucked from an unlucky pet, passed through the flea’s rudimentary digestive system, and deposited in the fur as flea poop) or any live fleas on the cat. Flea allergies are the most common cause of miliary eczema in cats. Other possible sources of allergens might include contact allergens, something the cat might walk on or roll in, or airborne pollens.

Diet might be another cause of allergies.

Since “miliary eczema” defines only the appearance of the rash, other clues to its cause may be found by noting where the rash occurs on the body. For example, if a rash has broken out near the ears, your vet may look for ear mites. Both bacterial and fungal skin infections may cause similar looking rashes. After questions about your cat’s environment, in particular about any changes or recent introductions to your home or yard, your veterinarian will try to pinpoint a precise cause. Sometimes a skin biopsy is recommended for definitive diagnosis. In some instances the underlying cause is never fully understood.

In Alfred’s case the rash appeared to be a response to an unknown allergen and a trial medication with an injectable steroid was administered. This kind of steroid is a glucocorticoid, not the anabolic steroids infamously abused by some athletes. Cats are somewhat unique here, being resistant to the effects that steroids have on dogs (and people) – they suffer far fewer side effects such as hunger and thirst, as a general rule, but they also respond less well to steroid medications in general. Most cats require injectable medication rather than oral steroid medication for allergies.

Alfred was most gracious about receiving an injection in his derriere and went home to convalesce. Overnight his owner reported that he seemed more comfortable, less restless. By day three Alfred was no longer over-grooming himself (it had crept up so slowly she hadn’t noticed his frantic licking increasing) and was eating at a leisurely pace, not jumping about as if suddenly pricked by something. Within the week the little scabs began to dry up and melt away, leaving smooth skin underneath. By the second week it was clear his fur was growing back in. Alfred was once again a gorgeous golden mini panther!

Alfred was lucky. As the effects of the medication wore off around week six there was no sign of recurrence of the rash. For many cats, if the owner is diligent about flea control their cat will remain clear skinned. For others, a combination of injectable or oral medication may be necessary year round to keep their cat comfortable.

Christine B. McFadden, DVM
drmc@mcmenagerie.com
Valley Animal Hospital, Merced

Foxtails

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Dr Mc Blog

 

March 10, 2015
At home I mark the seasons by watching the ever changing flowers along our roadsides and gracing our gardens. Spring brings the first forsythia flowers of bright yellow sunshine, the pink and white magnolia blossoms. At work I am waiting for the veterinary equivalent of that first harbinger of spring, the foxtail weed sticker.

It’s coming, that moment when I look at the chart of my next patient and read “cause for visit : foxtail in ear”.

The foxtail is the seed head of several weedy grasses. Sometimes called wild barley, several different grasses produce an awn or sticker that is pointed and barbed like a miniature badminton birdie. As your dog or cat brushes through the tall grasses the foxtail will catch in their fur, working in deeper by means of the pointed base.  Foxtails burrow deeply wherever they enter, travelling in one direction, prevented from backing out or falling out by their bushy tail.

The most common sites for foxtails to lodge in a pet are the ear and between the toes. The eyes, nose and vulva are also vulnerable, though foxtails can burrow into the skin anywhere, especially in thickly furred or matted animals.

Not only are the burrowing weeds painful, they can cause serious infections and abscesses wherever they enter the body. The sticker does not break down inside. It’s pointed end can burrow through ear drums, into eyeballs and travel up legs from the toes. Unless removed, a foxtail can eventually lead to death. Foxtails breathed into the nose can lodge there and run the risk of being inhaled into the lungs if not promptly removed. Foxtails eaten may escape the stomach and tunnel through the internal organs. Foxtails can kill.

Although you can’t always prevent contact between your pet and a foxtail, you can decrease the risk by taking the following steps :

  1. Don’t let your pet run through grassy fields or empty lots filled with weeds.
  2. Brush your pet after a trip outdoors, paying special attention to the face and feet. Gently separate each toe to look for tiny tan stickers between the feet.
  3. Pets with short, slick coats are not immune to picking up a foxtail, but there’s no denying that long silky hair or “feathers”, found in dogs like the Spaniel breeds or Golden Retrievers, attract the stickers. Thick, curly coats – think Poodles or Chow Chows – can also be foxtail magnets. These breeds may do better if their lower legs, toes and underbelly are trimmed short for the summer. A dog groomer can do this safely and easily for you. A “Summer Strip” that shears a long-haired dogs’ coat to an overall length of about ½ inch helps protect heavily coated dogs from picking up the stickers, with the added bonus of cooling your pet off during our hot summers.
  4. If possible, remove all weeds from your yard in the very early, green stage. Mow the weeds down, rake up and dispose. It is the dried seed head, in its bid to reproduce and spread far and wide, that detaches from the mother plant and hitches a ride on your pet to be carried to new fields. Elimination in the green stage is far easier to control and prevents further dispersal of seed heads. Foxtails are not readily air borne, but once dry “jump” off the plant onto whatever brushes against them.

Signs that might lead you to suspect that your pet has encountered a foxtail might include the following, especially if just after a walk or run :

  1. Your pet suddenly holds an ear down, shakes or rubs his head vigorously, crying out if you touch the ear.
  2. Repeated bursts of paroxysmal sneezing, so hard it looks like her nose might hit the floor.
  3. An ugly swelling between the toes, limping on one or more feet.

Most foxtails must be probed for and removed under anesthesia. Those lodged in the ear offer the sporting chance that, moving quickly enough, one can grab the offending sticker and be done with it. It’s the closest we come to a rodeo in small animal medicine. Given that the ear is awfully close to the teeth there can be a bit of a rush as the veterinarian eyes his or her prospective patient, calculating the odds of success.

Hmmm, the moment is here.” Check ears, foxtail”. The first patient this spring? A Newfoundland, the sort of dog that ought to live at the North Pole where there are no foxtails, and pick on someone his own size, like a polar bear. This dog looks to be about 150 pounds and growing by the second. Probably eats vets like they’re cheetos. And judging by his head tilt and red ear, it hurts, BAD.

We try the calm, business-as-usual approach. Let’s take a little peek here…. Nope. Next the hold-on-to-him, person on each end approach. Uhn-uh. Three people try to restrain his head. Help! Finally, a moment of stillness and we’ve got it! Quickly a quarter inch foxtail is extracted from the ear. Everyone takes a collective breath. The ear is cleaned and medicated. The Newfie is delighted, giving us doggie high fives with his nose which knock us into the wall. We all beam at each other. Success is sweet today. We know there will be more foxtail challenges tomorrow.

Christine B. McFadden, DVM
drmc@mcmenagerie.com
Valley Animal Hospital, Merced

Snap the Cat and the Broken Jaw

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Dr Mc Blog

October 22, 2014
His name was Snap. He had some Siamese in there somewhere because he had huge blue eyes and a white body with orange fur on his ears, tail and feet, a color we call “flame point”. A stunning kitten, he was all male : big, rangy body with long legs, on the prowl to explore my exam room as soon as he was set free from his carrier. Outgoing, slightly aggressive, he was not a cat to take well to indoor confinement. Fortunately Snap’s owners had a large yard and felt their kitten could grow up there safely. No animal is bred to live a life totally indoors, never seeing the sunshine.

Snap grew quickly, losing his first kitten baby tooth as he approached 4 months of age. Most kittens gain about one pound a month as they grow, but Snap gained more – he was going to be BIG! His owners reported that Snap, who got his name when he ran to them every time someone  snapped their fingers, had added fetching to his repertoire. Green beans were a favorite for some odd reason. Snap never ate the beans himself, but his family hated to be wasteful and found themselves researching new green bean recipes as they tried to keep up with his passion. Thanksgiving was the only time of year they could look forward to without guilt (add canned mushroom soup and French fried onions for a traditional casserole).

When Snap was neutered at about 6 months of age he was just losing his baby fang (canine) teeth and looked quite adorable. Neutering helped to suppress his urge to wander and would markedly decrease the likelihood of fights with other cats, which in turn could expose him to diseases like feline leukemia, feline AIDS (FIV, the Feline immunodeficiency virus, NOT infectious to people) or problems like abscesses. “Tomcats”, an old fashioned expression for unaltered or intact male cats can also develop “stud tail” – you don’t want this folks, however manly the name may sound. It’s a greasy patch that dirties the hair of the upper tail. Yech! And the urine of an intact male cat has a unique smell that can only be called a “stench”. It’s hard to draw breath in the same room. The good news is that one surgery and the smell, the greasy tail, and all just go away and you are left to enjoy your cat.

If the average cat weighs 10 pounds, Snap was out to prove that he was anything but average. By 2 years he was 14 pounds of lean muscle, extremely athletic and he enjoyed accompanying his owners on their nightly neighborhood walks. He was a powerful hunter of toy mice. He could jump from a standstill straight up ten feet. He talked frequently, expressing his disapproval if they were a little slow serving him breakfast. Snap had scheduled petting times, also, and showed his displeasure if one was missed by turning his back to the family when they finally came home.

But one night it was Snap who didn’t come home.

The following day we received a phone call that he had been found and was being rushed down to the hospital. And oh, the look of him made our collective stomachs drop. The beautiful fur was wet from mud and blood. His jaw hung crookedly and his nostrils were plugged with clots of blood. He was able to stand and even walk, though there were long scratches abrading one side of his body. It was obvious that Snap had been hit by a car, thrown through the air, and slid hard coming down. The broken face had been the site of impact.

Animals suffer from concussion the same as people. Snap’s pupils were pinpoint (meiotic) but he was responsive and even in a daze he was able to follow visual cues to show us he could still see. We started him on IV fluids and shock therapy and under gentle sedation assessed and cleaned his wounds and took x-rays of his skull. It was pretty bad. He had fractured the center of his lower jaw, so the right side of the mandible was split from the left. Worse, his upper jaw or maxilla had broken and the pieces shifted so that part of the roof of his mouth, the hard palate, was shoved upwards on the right, crunching into his nasal passage and sinus. Oddly, the soft tissue of the hard palate, though bruised, was intact. We were able to wire the lower jaw together on the spot. There wasn’t much we could do about the roof of his mouth but wait and see. All the teeth still overlapped correctly, so he could close his mouth properly. We inserted a feeding tube down his esophagus to the stomach and threaded it out the side of his neck to facilitate eating during recovery. And what a recovery. By day 3 his pupils were normal and his attitude improving. By day 5 he was eating soft food on his own, so IV fluids and feeding tubes were removed. Snap went home, returning to have the wire in his lower jaw removed 6 weeks later. Snap lived to be 15 years old. He never liked to have his mouth examined after all that and his face was a little crooked, but we knew he’d made it when he left a green bean on his owner’s pillow one night.

Christine McFadden, DVM

Valley Animal Hospital

Fleas Part Two : Is Zero Tolerance Possible?

/ Dr. Christine McFadden / Leave a comment

Dr Mc Blog

September 14, 2014
In my previous column on fleas we discussed the mode of action and the suffering and disease which fleas inflict upon dogs, cats, and occasionally people.  I stated that the tapeworm of dogs, Diplydium caninum, did not infect people. Dr. Jerry Menefee of Los Banos called to tell me how much he enjoyed my column and to gently correct me: there have been a few cases of human infestation with the dog tapeworm, called Diplydiasis, primarily when a young child or infant has actually eaten an infected flea (same as the dog or cat) and the worm develops in their intestine. There is rarely any sign of illness, just little tapeworm segments passing in the diaper! Treament for the child is the same as used for the dog, with excellent success. I could find 2 medical papers printed on separate cases, one published in 1993, another in 2003. Diplydiasis appears to be extremely rare (but oh dear, aren’t you running to check the calendar to see if you’re up to date on your flea control?); I shall expose the mysteries of “human tapeworm”, an entirely different group of worms, in a future column. Today we are exploring methods of flea prevention, critical in our area because, as noted in Part One, our local climate favors fleas – we are a flea hospitality haven!

If a pest has been around this long, there’s been plenty of time to try to get rid of it. Because some pets and people seem to attract more than their fair share of attention and get more bites, researchers tried changing the way skin smells. As it turns out, fleas are NOT put off by garlic or B vitamins, nor are they scared away by clove oil, tea tree oil, cinnamon, lemon, etc. Whether ingested or applied topically, these products only make you or your pet less socially acceptable to other like creatures. Not recommended. It has been theorized that some dogs and cats are better able to groom out fleas with their teeth, so appear to be flea free and escape the ugly monikker “fleabag”.

So we’re talking pesticides to control pests. Thirty years ago we used flea powders, sprays and flea collars, all powered by chemicals in the carbamate and organophosphate family of pesticides. This group of chemicals were not at all selective to fleas – used improperly, they could poison your pet and you. It’s what we had (our “zero”), and we were desperate. The collars worked sometimes on very small pets, since they only covered the neck. Powders would do the job, if you could stand the dust all over your pets’ coat. Sprays were very effective, but required near daily application because as soon as your dog rolled in the grass they were lost in the dew. Forget spraying a cat- one puff and that cat was in the next county. Dips were also effective, though again, none lasted two weeks as dogs roll in the grass…..removing the substance. Most flea dips were toxic to cats, so if applied incorrectly to a cat it would die unless emergency treatment was sought. I spent hours counselling on time consuming methods of flea control that no one thanked me for.

Pest control companies were kept busy spraying yards and houses in an effort to control the flea in the environment. One product, malathion, a mainstay in pest control years ago, to overcome flea resistance had to be sprayed over your entire yard every 10 days to have any efficacy at all according to UC Davis! DIY (do-it-yourself) friendly, it was not. Again, clients were grumpy to receive this news. Today there are a rotating group of pesticides used by pest control companies to fight the flea and prevent resistant strains from developing. Environmental control in the yard is very important; even if your own pet never leaves home, wildlife and roaming pets will almost certainly introduce fleas. Your pet does not live in a bubble!

And then a new generation of medications were discovered. Wonderstuff. Applied topically to the skin of the pets neck or given as a chewey pill, and only ONCE a month, this stuff is amazing! This stuff works! Several products exist for both dogs and cats, all kill fleas within 12 hours of contact with the pet. Reinfesting fleas, flea eggs and larvae in the environment are killed following contact with the treated dog or cat. If you do no environmental control, expect it to take about 90 days to clean up your flea problem. Some of the chemical names we’ll discuss are imidacloprid, spinosad, fipronil and afoxolaner. Ahhh, what’s in a name? Tongue twisters all – the brand names are easier. Curiously, most of these products weren’t developed specifically for your pet.

Spinosad is a pesticide developed to safeguard our food supply, used commercially on fruit and vegetable crops. Spinosad is considered an “organic” insecticide found in a bacteria from sugar cane. Spinosad is considered a natural product, given a Class B designation for pregnant women: No evidence of risk in humans.  It is used on people in medication for human head lice. On dogs and cats it has been found effective primarily against only the flea.

Imidacloprid, another flea fighter available for dogs and cats, is also used on crops and home back yards. Acting as a neurotoxin to insects, when used IN THE YARD, it is a systemic chemical that is taken up by plant roots to its leaves, where sucking/chewing insects will ingest it and die. There has been some question if imidacloprid may adversely affect honeybees (colony collapse disorder), though bees do NOT chew flower or plant parts.

Fipronil is an insecticide that affects the nervous system of insects via pathways that don’t exist in mammals. A broad spectrum insecticide, it is highly toxic to fish and bees. It is widely used for protection of rice crops, field corn, and golf course lawn care. Humans can prevent tick bites by applying it (as directed) to their ankles and wrists. Fipronil can kill rabbits – do NOT use on rabbits for flea control! That said, for years the State of Hawaii has required the veterinarian-only application of Fipronil, specifically, to all dogs and cats 10 days prior to travelling to the state. Fipronil has been widely questioned in honeybee colony collapse disorder.

Afoxolaner is in the newest class of chemical, this one without apparent crop/plant use. It’s sole use is flea AND tick control in dogs. It’s a new isoxazoline parasiticide (I had to read that, why not you?) – its’ claim to fame is that it controls both fleas and ticks via an oral medication for an entire month. Tick control has always been difficult in dogs, and there are many serious tick-borne diseases. The class of isoxazaline drugs may be a breakthrough for livestock that suffer from both ticks and tick borne diseases worldwide.

The flea is here to stay. With considered use of the available products you may be able to control you and your pets’ exposure to fleas and the consequences of flea bites. We aren’t talking eradication. You are not expected to reach Zero.  As for your children, your home and yard, the truth is that flea control on the pet is not deemed of sufficient quantity to add to or create environmental issues at this time. Most of the products we’ve discussed are carried home from Garden Centers and Big Box stores by the quart and gallon weekly as yard and grass treatment for indiscriminate use and disposal by the homeowner. These products all appear much safer for mammals – you, your children, your dogs and cats – than previous options, and every year newer products are developed as we all cope to care for each other and our earth.

Christine B. McFadden, DVM
Valley Animal Hospital, Merced
drmc@mcmenagerie.com

The Flea and Zero Tolerance : Part One

/ Dr. Christine McFadden / Leave a comment

 

Dr Mc Blog

September 7, 2014
One of the more curious things worth pondering is the realization that “zero” is only as low as your ability to measure something, any given parameter, to zero. How much of a given substance is found in your food, how much radioactive activity persists in the ground, how fast a ball flies from the pitchers hand is all measured by a variety of instruments created by man and only as accurate as the instrument currently in use. As technology advances we are able to detect smaller amounts, faster movement, ever tinier invisible particles – and our previously accepted “zero level” decreases accordingly. Fascinating!

Enter the flea, ctenocephalides felis, the common flea of both dogs and cats despite its’ Latin name. The flea has been with us for centuries. A small wing-less creature, it can safely be described as a parasite to most of us, if we accept the definition of parasite to be an organism that exploits or lives off another organism while contributing nothing to its wellbeing. Why do we care what this little insect gets up to in its daily life?

For starters, fleas rarely appear to be doing good deeds. Once an adult flea has pounced upon a dog or cat, it will feast upon its blood (blood meal). In very small, young, or very ill pets enough fleas can together literally suck them dry of their lifeblood, leading to severe flea anemia and death.

While taking that blood meal, the flea actually injects its mouthpart or proboscis into your pets’ flesh which also introduces its saliva. Not only does your pet feel little flea feet crawling all over its skin, it can feel the sting of the bite and worse, develop an allergic reaction to the flea saliva, which will make him itch and break out in a rash. The ultimate reaction is a pet so allergic that they scratch off their hair, their skin, and develop oozing, painful sores called “hot spots”. Medical attention is required to get these under control quickly. Your veterinarian can literally start turning this condition around in hours! Hot spot and other allergy medication does not take days or weeks to work!

Which came first, the flea or the tapeworm egg? In a twist of fate, the tapeworm egg of Diplydium caninum lays on the ground or carpet. A flea larvae MUST eat it for the tapeworm to continue to develop. So the flea larvae eats the tapeworm egg. When the flea bites your pet, the tapeworm larvae is spit out on their coat. Your dog or cat turns to lick at the stinging flea bite and ingests the tapeworm larvae, which is swallowed and grows up inside your pet to become long ribbons of worm segments looped together in the intestine. Often these live segments break apart and are passed in pet feces. You may literally see wiggling flat worms (proglottids to you science types) in pet droppings or even around your pets’ tail. Once dried, they may look like rice or dried sesame seeds. They do not pass daily, so if left untreated don’t be fooled that they’ve naturally gone away if you don’t see them for a week or two. They’re still there.

Ok, so far we’ve discussed fleas on cats and dogs :
anemia, even death;
creepy crawly flea feet running on them;
allergies, hot spots;
tapeworms.

What about people?Some people are bitten by fleas. The bites can be painful, and may cause an allergic skin reaction. The kind of tapeworm, Diplydium caninum, that dogs and cats carry is NOT the tapeworm you hear people can get (they must eat a flea to get this kind of tapeworm – highly uncommon).

But there IS a zoonotic (spread from animals to people) infection fleas CAN transmit: murine typhus, a rickettsial bacteria that may be harbored in feral or pet cats, opposums, and other animals. Murine typhus is an under-discussed suburban vector-borne zoonotic  disease. In the United States California, Texas and Hawaii have had outbreaks. In California, the southern areas of the state are vulnerable. Orange County reported two (2) cases of murine typhus in 2012. When one case was reported in a young girl the county trapped feral cats and opposums near her elementary school searching for the probable source of contagion.  This disease usually responds well to antibiotics and people are easily cured. However, it is uncommon and initial diagnosis can be difficult. The primary recommendation to decrease the risk of murine typhus? 1) Do not feed feral or wild animals near your home. 2) Apply flea prevention/control products to your pets.

Which brings us to the crux of this article. How to prevent fleas on your pets, in your home, on you? Because if you live in Merced County, we provide everything a flea needs to flourish with our long seasons of warm weather and mild winters. We are a flea hospitality haven! Please tune in for Fleas: Part Two : Searching for Ground Zero

Christine B. McFadden, DVM
Valley Animal Hospital, Merced
drmc@mcmenagerie.com